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Description

This makes GSH replacement therapy attractive, because it has the potential to interfere with the expression of the integrated HIV genome, a site that is not attacked by the currently employed antiretrovirals (AZT, ddl, ddC, D4T)

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Association of UCP2 866 G/A polymorphism with chronic inflammatory diseases

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Identification of novel TGF /SMAD gene targets in dermal fibroblasts using a combined cDNA microarray/promoter transactivation approach

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