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Methods: Two immunocompetent HCC models were used: an endogenous genetically heterogeneous HCC model created via hydrodynamic tail vein injection of a 10-oncogene cDNA library, and a subcutaneous syngeneic model using the Hep55.1c cell line

Our study reinforces the emerging paradigm that necroptosis is not merely a backup death pathway, but rather a central and targetable mechanism in cancer control