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The potential of these nanoparticles lies in their ability to facilitate the passage of medications through the blood-brain barrier, thereby enhancing the effective delivery of treatments for neurological conditions (154, 155)

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Such clinical syndrome exacerbates the muscle loss and fosters sarcopenia progression

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The mechanism by which toxicity results is complex, but is believed to involve reaction between unconjugated NAPQI and critical proteins as well as increased susceptibility to oxidative stress caused by the depletion of glutathione

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