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Description

129 Exogenous SAM can inhibit HCC development by recovering the normal level of SAM

They showed that IGF-1 delays the onset of behavioural symptoms and sustains life to a greater degree than GDNF

Journal of Central Nervous System Disease

It supports detoxification, immune function, and cellular repair

Conversely, when cellular energy demands increase, LDs can be selectively degraded through autophagy-dependent lipophagy to release FAs for beta-oxidation and ATP production [93, 94]