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Notably, Homocysteine can react and modify specific proteins, resulting in neo-antigens development and autoimmunity development (Lazzerini et al

Indirect evidence for this possibility was provided by the high expression levels of the SA-induced gene PR1 in untreated and BSO+GSH-treated sym33-3 plants relative to other genotypes (Figure 7)

Ferroptosis, a newly characterized form of cell death in Parkinsons disease that is regulated by PKC

Programming bacteria response to intracellular nanoassemblies To equip the bacteria with the means to expel the NPs, we engineered them with a regulatory gene circuit, which is activated upon oxidative stress

Its mechanism of action involves the olfactory and trigeminal nervous systems, which originate from the olfactory neuroepithelium in the nasal cavity and terminate in the brain