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Preclinical animal research has suggested that the progression of diabetic cardiomyopathy (DCM) is affected by the presence of reactive oxygen species (ROS)

338 Similar decreases in astrocytic expression of glutamate transports are characteristic for animal models of epilepsy, 339,340 and genetic ablation of EAAT1/2 triggers seizures

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These effects, induced by arecaidine, appear to be mediated by ROS production as the co-incubation with the antioxidant N -acetyl-l-cysteine decreases ROS levels and the apoptotic index in U87MG and U251MG GBM cell lines

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