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This is one mechanistic reason IV and parenteral administration have shown some tissue-level effects in animal studies

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Gout, a painful arthritic condition caused by the deposition of urate crystals in joints, is one of the most widely recognized consequences of elevated UA levels

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TREX1-deficient mice develop aberrant interferogenic responses and features of lupus owing to the cytoplasmic accumulation of endogenous nucleic acids and chronic activation of the TBK1-dependent DNA-sensing pathway (6870)