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The above research results suggest that m6A modification may be closely related to the occurrence and development of ferroptosis in AD and requires further in-depth research for verification

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These findings suggest that it is possible to regulate ferroptosis through artificial intervention, which will help develop targeted therapies related to ferroptosis

Immunofluorescence staining was performed to assess macrophage polarization, with CD86 serving as a marker for pro-inflammatory M1 macrophages and CD206 for anti-inflammatory M2 macrophages

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