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This is followed by the addition of glycine to the C-terminus of gamma-glutamylcysteine via the enzyme GSH synthetase to form GSH

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Since it has been previously reported that in endothelial cells (ECs), eNOS activation can be induced via Ca-dependent dimerization and Akt-dependent phosphorylation, ultimately enhancing NO production and vascular function [71], we sought to investigate whether a similar mechanism is involved in our model

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(1988) Blood, 71(2): 335-343