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The ensuing energy shortage compels immune cells to resort to less efficient energy production pathways, which in turn may lead to the generation of additional inflammatory mediators, thereby worsening mitochondrial dysfunction in peripheral and central cells (142)

synthetic analogs may be used in conjunction with mineralocorticoids where applicable

FUNDC1/USP15/Drp1 ameliorated TNF--induced pulmonary artery endothelial cell proliferation by regulating mitochondrial dynamics

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