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For instance, CHF is caused by impaired cardiac pump function, which leads to fluid retention, which prolongs disease progression and hospital readmission (132, 133)

Excessive oxidation of GDH leads to a decrease of glutamate in hepatocytes, which in turn reduces the production of N-acetylglutamate (NAG), an allosteric activator of carbamoyl phosphate synthetase (CPS)

for example, multinodular goiter and thyroid adenomas are associated with these deficiencies [1]

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As a thiol-containing compound, GSH may regulate the redox sites of several ionotropic receptors and ion channels, altering their functional characteristics (Varga et al., 1997) through the glutamatergic receptors NMDA and -amino-3-hydroxy-5-methylisoxazole-4-propionic acid (AMPA)