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This interaction was specific, as the NINJ1 knockdown significantly reduced PLA signal and neither NINJ1 nor xCT antibody alone led to significant PLA signals (Fig

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p16 expression is weak in healthy cells, but in senescent cells, p16 binds to and inhibits cell cycle protein-dependent kinase 4/6 (CDK4/6) activity, thereby promoting retinoblastoma (RB)-dependent cell cycle arrest [29,30,31]

Better insulin sensitivity means more efficient nutrient partitioning, more glucose directed to muscle rather than fat, and better preservation of lean mass during caloric deficit