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& Vande Velde, C

Conversely, cuproptosis, a copper-induced cell death triggered by the accumulation of Cu in mitochondria, leads to the aggregation of lipoylated dihydrolipoamide S-acetyltransferase (DLAT) and a loss of Fe-S protein, which are associated with energy formation in the tricarboxylic acid cycle (TCA), leading to proteotoxic cellular stress, eventually leading to cell death [7]

While generally considered safe, its important to consult with a qualified healthcare provider, especially if you have underlying health conditions or are taking other medications

However, the connection between iron metabolism and T2D, along with its consequences, remained ambiguous until the identification of ferroptosis

Moreover, L-Carnitine supplementation has been suggested to prevent protein catabolism [47], which is corroborated by the increase in mTOR expression observed by the end of the study