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What is increasingly emerging from the literature is that, although it is traditionally considered an extracellular pathogen, the meningococcus engages in complex interactions with host cells in the intracellular microenvironment

These effects can lead to: Reduced dietary intake of B12-rich foods (meat, fish, dairy, eggs) Altered eating patterns with potential nutritional inadequacy Reduced nutritional intake if gastrointestinal symptoms are severe or prolonged Vitamin B12 deficiency can manifest with significant clinical consequences, including macrocytic anaemia, peripheral neuropathy, cognitive impairment, fatigue, and mood disturbances

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R.RechR.Ollivault-ShiflettM.Esteve-GasentM

doi: 10.1186/s12967-020-02419-4 262 MackayA