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Curcumin, a well-known active substance extracted from the turmeric rhizome, destabilizes endoplasmic reticulum, and secondarily lysosomes, thereby increasing mitophagy/autophagy and subsequent apoptosis

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Isocitrate dehydrogenase-wildtype glioma adapts toward mutant phenotypes and enhanced therapy sensitivity under D-2-hydroxyglutarate exposure

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Soluble uric acid can interact with TLR2 agonists to decrease M2 marker expression and increase proinflammatory gene transcription, shifting macrophages toward an M1-like phenotype (120)