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Recently, active STING signaling has been linked to the survival of chromosomally unstable cancers in an IL-6 dependent manner involving both NF-B and signal transducer and activator of transcription 3 (STAT3) activation (226)

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No significant increase in ferritin was observed by treatment with the proteasome inhibitor MG132, suggesting that the proteasome is not involved in ferritin degradation during CSE exposure (Fig