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The cancer driver genes IDH1/2, JARID1C/KDM5C, and UTX/KDM6A: crosstalk between histone demethylation and hypoxic reprogramming in cancer metabolism

Furthermore, in the presence or absence of GSH synthesis, tumours have an adequate supply of upstream metabolites (that is, cysteine) to sustain growth

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In this particular case, the patient also had beta-thalassemia, a condition that increases iron load and oxidative stress, accelerating cataract formation (59)