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As it has been claimed by Blom and colleagues, cJun activation in Schwann cells is JNKindependent in the case of peripheral nerve injury and the authors have proposed that other MAPKs are also capable of stimulating cJun (Parkinson et al., 2008) and controlled the phenotypical changes in markers expression patterns inside the Schwann cells as they dedifferentiate, also controlled neuronal survival, promoted axonal growth and myelin degradation in cooperation with the macrophages during peripheral nerve regeneration (Lepp et al., 1998