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when it is inhibited, neurons quickly restore glutamate neurotransmission by an ill-defined endogenous mechanism, suggesting that neurons might be making use of a storage buffer of glutamate (15)

However, NOM consists of relatively large organic molecules, with varying structures depending on their origin, containing a multitude of functional groups and it is sometimes difficult to interpret results from experimental work involving NOM

Translating these preclinical findings into clinical applications requires validation of exact molecular mechanisms and dose-response relationships, particularly for H 2 S, to develop targeted therapies for CRC

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J., Hatzivassiliou, G