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Here, we focus on the molecular mechanisms that support the elaboration of anticancer therapies that modulate the production and scavenging of ROS and, in particular, on the opportunities raised by their ability to induce cell death upon exceeding a threshold level

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Since, 9 -THC does not produce maximal stimulation of the above receptors, the background of such changes could result from blocking endogenous full agonist, i.e., anandamide from binding to CB 1 receptor, especially, when the endocannabinoid tone is high (e.g., obesity) ( 9 -THC in the prevention and treatment of obesity requires further investigation