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Notably, the liver, a primary organ for threonines initial catabolism, demonstrated a notable increase in SDS activity, facilitating threonines conversion into propionyl-CoA across all detected organs during fasting, likely due to the inter-organ cross-talk mediated by downstream metabolites, although muscle SDS activity remained unchanged

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All the tested membrane proteins displayed higher expression levels with TGP compared to GFPs in both prokaryotic and eukaryotic systems

Modulatory effect of long-term treatment with escitalopram and clonazepam on the expression of anxiety-related neuropeptides: neuromedin U, neuropeptide S and their receptors in the rat brain