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Animal models have further supported the role of glutathione in lung injury, with glutathione deficient mice having impaired tolerance of oxidative stress, and abnormal early lung development 43

doi: 10.3389/fimmu.2022.881427 83 GrabowskiMMSankeyEWRyanKJChongsathidkietPLorreySJWilkinsonDSet al

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Consistent with the results from germline XKR8 KO mice challenged with LPS, neutrophil-specific XKR8 KO mice displayed significantly reduced NET formation in the lungs, lower levels of extracellular dsDNA in BALF and mitigated lung injury, despite similar levels of neutrophil infiltration following LPS challenge (Fig