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SLC7A11 disruption in PDAC cell lines strongly affects their amino acid and redox balance, and thus suppresses in vitro and delays in vivo their proliferative phenotype ( in vivo SLC7A11 knock out PDAC cells grew normally

Pre-treatment of MSCs with growth factors such as FGF-2, bone morphogenetic protein-2 (BMP-2), and insulin-like growth factor-1 (IGF-1) enhanced the expression of cardiac transcription factors, including GATA-4 and NKx-2.5, promoting cardiomyogenic differentiation and leading to improved therapeutic outcomes in myocardial injury [44]

The abnormal functions of mitochondria in cancer cells result in increased mitochondrial membrane potential (hyperpolarization) and excessive ROS production