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Loss of the capacity for homothallic self-mating could increase the frequency of out-crossing and this may be beneficial to some species under pressure from their environment or host 45 such as the pathogenic species in this group

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While CBP/p300 is a well-validated direct interaction partner of NRF2 with prominent functions at enhancers, we report that this interaction is not required for NRF2-dependent NSCLC cell growth, indicating that NRF2 can sustain sufficient transcriptional activity in the absence of CBP/p300 coactivation