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M.GuigasB.HormanS.DentinR.BertrandL.HueL.et al (2006)

The development of more potent GlyT1 inhibitors was initially inspired by structural modifications of sarcosine, particularly through the introduction of bulky hydrophobic substituents at the nitrogen atom, an approach guided by emerging structural and pharmacological insights into GlyT1 and its relationship to other members of the SLC6 transporter family

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Another is that erastin restrains systemic X C activity and causes the depletion of GSH, thereby inactivating the GPX4 enzyme, which then induces ROS formation, selectively inducing ferroptosis in HRASV12 mutant BJeLR cells (Dixon et al., 2012)