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3.3 From interface to intracellular redox availability The core mechanistic question is not simply whether phenolics can be loaded, but whether interfacial design yields the intended intracellular redox kinetics (in terms of timing, dose, and compartment)

Most intrinsic HAs are inherited disorders, but paroxysmal nocturnal hemoglobinuria is exceptional [7]

Additionally, research indicates that Safflower yellow, a major active constituent of Carthamus tinctorius L., can influence HCC progression by modulating the GM and enhancing hepatic immune infiltration [16]